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Mechanistically, SAHA pretreatment resulted in the decrease of histone H3 acetylated at lysine 9 (H3K9) of TLR4 gene and inhibited TLR4/MyD88 signaling pathway, finally protected from KA-induced brain damage [150]

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It involves an identified patient, a licensed prescriber, a clinical rationale, pharmacy accountability, and the possibility of follow-up and adverse-event evaluation

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