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Exenatide restores GLP-1R expression and reduces mucin production in COPD airway cells by restoring FOXA2 function through activation of GLP-1R-peroxisome proliferator-activated receptor gamma (PPAR-)-phosphatase and tension homolog (PTEN) signaling, thereby increasing the ectopic and efficacy of other drugs in COPD and reducing the high morbidity and mortality induced by excess mucus