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Homocysteine induces melanocytes apoptosis via PERK-eIF2-CHOP pathway in vitiligo

Although GSH is not alone responsible for these protective mechanisms, the hepatic production of GSH seems to be essential to survival, with knockout mice dying within months secondary to inflammation and hepatic steatosis resulting in liver failure [21]

2017;70:e39-e110

Having one or more of these polymorphisms is associated with an increased risk of certain diseases, especially when impacted by environmental triggers such as pollution, smoking, heavy metals, and other toxins