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The observed reduction in GSSG levels reflects a compensatory regulation of redox balance, suggesting a potential self-protective mechanism that may play a crucial role in maintaining neurological homeostasis [48, 49]

Our analysis of The Cancer Genome Atlas (TCGA) human Liver Hepatocellular Carcinoma (LIHC) dataset confirmed that key genes involved in SAA metabolism, including MAT1A , BHMT , CBS , CTH , and CDO1 , are suppressed in HCC compared to normal liver (Supplementary Fig