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mansoni histone deacetylase 8, was not labeled with or without NAPDH

Elevated DNL activity not only promotes hepatic steatosis but may also contribute to hypertriglyceridemia, as fatty acids such as palmitate exhibit pro-inflammatory and pro-apoptotic effects [64, 65]

These results suggest that EAM-induced myocardial injury resulted in the release of H-FABP into the circulation, disrupted fatty acid transport, decreased -oxidation, and enhanced anaerobic glycolysis

In a second approach, organoids were stimulated with low concentrations of TNF/IFN and LPS for 48h to determine the effect on epithelial differentiation