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Previously, colchicine has been shown to provide anti-oxidative benefits [24, 25, 30], attenuate oxidative stress-induced DNA damage, impede pyroptosis and cellular senescence in endothelial cells [24, 26, 27]

139 Initially, it was hypothesized that the loss or reduction of ABCG2-mediated renal urate secretion would result in increased renal urate reabsorption, as diminished renal excretion is typically considered the primary mechanism of hyperuricemia in most gout patients

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